The latest in SRFs progress, research, & growth.
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Lifespan.io (Life Extension Advocacy Foundation) and SENS Research Foundation are excited to announce the completion of their merger, forming a new 501(c)(3) non-profit.
Lifespan Research Institute (“LRI”) will develop, promote, and ensure widespread access to regenerative medicine solutions targeting the disabilities and diseases of aging, combining direct research efforts with robust education, affiliation, and outreach programs. The newly merged organization will focus on two core areas: Research and Outreach.
SENS Research Foundation is seeking volunteers for blood donations.
Your participation will help advance our understanding of the immune system and mitochondrial biology in aging.
SENS Research Foundation, dedicated to advancing rejuvenation biotechnologies, has recently begun subleasing lab space in the heart of Silicon Valley.
The sublease includes access to an extensive list of laboratory equipment, a break room, conference rooms, and much more.
Watch SRF Science Writer Michael Rae‘s presentation Damage Repair is the Superior Approach Against Aging.
SRF is a proud sponsor of the Phaedon Institute’s Seno-Therapeutics Summit 2023. Dr. Amit Sharma spoke at the Summit – his presentation was entitled, “Next-generation Senotherapeutics: From harnessing the immune system to targeting newer vulnerabilities of senescent cells.”
The summit was hosted by the Buck Institute For Research On Aging in Novato, CA. Entitled Seno-Therapeutics: Past, Present & Future of the Science of Cellular Senescence and Therapeutics, the summit was held November 7-8, 2023.
Thank you to our Director of Academic Affairs, Dr. Lilli Fishman, for participating in the National Academies of Sciences, Engineering, and Medicine GUIRR unit’s Industry Forum.
Thank you to Kriti Bhardwaj and Kartik Sundaram for presenting their final presentation as part of the 2024 SRF Post-baccalaureate fellowship and graduate internship programs!
Welcome to our new Post-Baccalaureates for the 2024=2025 year!
Matt Kaeberlein, Ph.D., CEO of Optispan, Adjunct Professor of Oral Health Sciences at UW, and Co-Director of the Dog Aging Project presented Longevity Science: From Molecules to Pets to Clinical Practice.
A supporter asks: Everyone knows that mitochondrial free radicals are a key driver of aging, and antioxidants don’t seem to offer any protection. Birds are supposed to have very clean-burning mitochondria, so should you maybe try to cut them off at the source by re-engineering our mitochondria to be more like those of birds?
A supporter asks if “backing up” copies of the mitochondrially-encoded genes in the nucleus is really viable, granted free radical damage in the nucleus. We emphasize the many additional ways that the nuclear copies will be safer than the mitochondrial originals, that the “backup copies” can be backed up again, and how they and additional strategies will buy us time for even better solutions.
Aging muscles lose strength above and beyond what would be expected from the mere loss of muscle mass. Accordingly, many drugs have been shown to stimulate muscle growth in older people, but the increased muscle mass consistently fails to translate into increased strength and physical function. To let people live independent lives for longer, we need damage-repair longevity therapeutics to repair the cellular and molecular damage that makes aging muscle dysfunctional.
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